◇ Compound Reference
Ghrelin Mimetic · 10mg
Selective growth hormone secretagogue. Minimal impact on cortisol or prolactin. Standard pairing with CJC-1295 in GH-optimization stacks.
Ghrelin Mimetic · 10mg per vial · dispensed after a physician review.
Ipamorelin is a 5-amino-acid synthetic GHRP (Growth Hormone Releasing Peptide) and selective ghrelin receptor agonist. Distinguished from earlier GHRPs by minimal effect on cortisol or prolactin, which makes it a preferred pairing partner for GHRH analogs.
Selective ghrelin receptor (GHS-R) agonism. Stimulates GH release through a different pathway than GHRH analogs, creating synergy when paired.
Minimal off-target effects vs. earlier GHRPs.
GHRH / GHRP secretagogues - Tesamorelin, CJC-1295, Ipamorelin, V-G1
These protocols gently restore your body’s own pulsing release of growth hormone rather than replacing it. Because growth hormone also affects blood sugar, monitoring confirms two things: that it is working, and that it is doing so safely.
Incremental approach. Vivre starts below the standard dose and advances only on tolerance. This monitoring is the safety scaffold for that conservative approach.
Educational only - not medical advice or a dosing instruction. Your physician orders the tests, sets your dose, interprets results, and decides if a protocol is right for you. See the Informed Consent & Disclaimer for full terms.
| Class | Selective ghrelin-receptor (GHS-R) agonist / GH secretagogue |
| Mechanism | GHS-R agonism; stimulates GH with relative selectivity (limited cortisol/prolactin effect) |
| Terminal half-life | Short (~2 hours order) |
| Metabolism | Peptidase degradation |
| Evidence base | Moderate; selectivity profile relatively well-described |
| Regulatory status | Not an approved therapy; physician-supervised use only |
Foundational preclinical and early clinical study establishing Ipamorelin's selectivity profile. In swine, no GH secretagogue tested affected FSH, LH, PRL, or TSH plasma levels - but GHRP-6 and GHRP-2 elevated ACTH and cortisol meaningfully, while Ipamorelin did not (even at doses 200-fold above the ED50 for GH release). This selectivity is the principal clinical reason Ipamorelin is preferred in modern GH-axis stacks over earlier GHRPs.
View on publisher ↗Preclinical rat study examining downstream skeletal effects of Ipamorelin-driven GH release. Demonstrated dose-dependent increases in longitudinal bone growth via the GH/IGF-1 axis. Important methodology framing: rat model with pre-pubertal skeletal growth - does not translate directly to adult human use, but supports the IGF-1 elevation mechanism that anchors body-composition and recovery applications.
View on publisher ↗Ipamorelin is verified and lot-tested, dispensed following a Biological Audit. The Audit is complimentary for the Batch 001 cohort.
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